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Data are presented which strongly indicate that the locus occupied by the mutant ws lies to the left of, or on the same locus of apricot (w^(α)). The fact that w shows a non-suppressor effect in combination with zeste as a typical phenomenon in mutants of sites 1, 2, and 3 of the white locus, also supports that W is a mutant at or left of apricot (site 3). A "model" for the genetic fine structure of the mutants (w^(2m), w^(zl), and w^(z)) is proposed and discussed to account for the mutability among those three mutants. Analysis of ws leads to the hypo-thesis that it is the result of an inversion (sites 3 and 4) at the white locus, by two mechanisms which are discussed briefly here.
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